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He presence of higher transverse diameter and temperature in the injured area together with the higher infiltration of the inflammatory cells in the injured area of the treated lesions during the first 14 DPI, suggest that a higher inflammatory reaction has commenced, the healing response has been motivated by the collagen implant and the metabolism of the injured area has increased. At earlier st
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E and the mechanism by which they might act in oestrogen-responsive breast cancer is unknown. Similarly, the signal transduction pathways involved in the induction of anoikis and in anoikis resistance of breast cancer cells have not been determined. There have been a few studies that purport to examine the effects of the IGF signal transduction pathway upon anoikis breast cancer cells. However, ca
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Ls from the extracellular matrix initiates programmed cell death by a process termed anoikis. Malignant cells must acquire anoikis resistance to leave the primary tumour and metastasise. Multiple signal transduction pathways can activate anoikis and confer anoikis resistance, but these are not understood in breast cancer. Methods: Models for anoikis of oestrogen-responsive breast cancer cells were
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